News and Announcements
Read the latest news from Northwestern University Feinberg School of Medicine’s Department of Pharmacology and learn more about our faculty’s latest achievements, awards and honors.
Northwestern Medicine scientists have characterized several variants of the gene GRIK2 that cause nonsyndromic neurodevelopmental disorder.
Circulating tumor cells use the surface protein ICAM1 to strengthen stem cell programs and facilitate formation of tumor cell clusters, which can travel from primary tumors to other organs in the body.
Gabriel Rocklin, PhD, is an assistant professor of Pharmacology and a member of the Robert H. Lurie Comprehensive Cancer Center of Northwestern University. He is also a faculty member at Northwestern’s Center for Synthetic Biology, where his lab develops high-throughput methods for protein biophysics and protein design, with a focus on protein therapeutics.
In collaboration with Northwestern Medicine investigators, an international multi-center study has identified genetic factors associated with SARS-CoV-2 infection and COVID-19 severity.
Feinberg’s Department of Pharmacology has expanded in size, faculty, and research focus, and is looking ahead to future growth in the field.
Scientists discovered a previously unknown receptor site in a voltage-gated sodium channel, suggesting that a commonly-used cancer drug could be used to target this channel and relieve pain.
For the first time, Northwestern Medicine scientists have characterized how a genetic mutation associated with pediatric epilepsy affects neuron activity.
A team led by Northwestern Medicine investigators has identified a novel molecular target that may improve the efficacy of current treatments for triple-negative breast cancer.
Calcium channels commonly found in immune cells are also present in the brain and regulate synaptic plasticity, according to a recently published study.
Northwestern Medicine investigators have identified a novel “gate latch” mechanism within the Orai1 ion channel that is essential for proper activation of the immune system.
Northwestern scientists have determined how two protein mutations responsible for the impaired motor function in Parkinson’s disease independently disrupt neuron activity.
A cell-surface protein is essential for proper microcircuit function in the brain, according to a study published in Nature Communications.